Studies on Renal Disorders (Hardback)

Edité par Humana Press Inc., 2010
ISBN 10: 1607618567 / ISBN 13: 9781607618560
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Language: English . Brand New Book. Conditions such as oxidative stress and hypoxia, which have a generalized impact on the oxygen metabolism, have been implicated in the genesis of kidney disease. This means that deepening our understanding of the pathobiology of oxygen metabolism in such diseases could be a fruitful path towards tangible clinical benefits. Studies in Renal Disorder collects reviews from leading researchers and clinical scientists working in exactly this field, providing an overview of the latest advances. The causal role of impaired oxygen metabolism in kidney disease has numerous clinical implications. It affects our understanding of the therapeutic benefits accruing from anti-hypertensive agents; the way we control hyperglycemia/hyperinsulinemia and hyperlipidemia; and our use of dietary approaches to the correction of obesity. The defensive mechanisms against oxidative stress, such as the Nrf2-Keap1 system, and hypoxia, such as the PHD-HIF system, have recently been explored in various cells, including kidney cells. These mechanisms include intracellular sensors for oxidative stress and hypoxia. This means that novel approaches targeting these sensors may offer clinical benefits in kidney disease in which oxidative stress and/or hypoxia is a final, common pathway. N° de réf. du libraire LIB9781607618560

A propos du livre :

Synopsis : Conditions such as oxidative stress and hypoxia, which have a generalized impact on the oxygen metabolism, have been implicated in the genesis of kidney disease. This means that deepening our understanding of the pathobiology of oxygen metabolism in such diseases could be a fruitful path towards tangible clinical benefits. Studies in Renal Disorder collects reviews from leading researchers and clinical scientists working in exactly this field, providing an overview of the latest advances. The causal role of impaired oxygen metabolism in kidney disease has numerous clinical implications. It affects our understanding of the therapeutic benefits accruing from anti-hypertensive agents; the way we control hyperglycemia/hyperinsulinemia and hyperlipidemia; and our use of dietary approaches to the correction of obesity. The defensive mechanisms against oxidative stress, such as the Nrf2-Keap1 system, and hypoxia, such as the PHD-HIF system, have recently been explored in various cells, including kidney cells. These mechanisms include intracellular sensors for oxidative stress and hypoxia. This means that novel approaches targeting these sensors may offer clinical benefits in kidney disease in which oxidative stress and/or hypoxia is a final, common pathway.

Review:

From the reviews:

“This book is a collection of reviews on derangements of oxygen metabolism in kidney diseases. ... addressed to students (mostly postgraduate), researchers, and academic clinicians ... . I liked the organization of each chapter with an abstract, key words, introduction, and conclusion as well as extensive, up-to-date, relevant references. ... This is the only book that I am aware of that is an extensive, updated review of abnormal oxygen metabolism in a large number of pathologic renal diseases.” (Mony Fraer, Doody’s Review Service, July, 2011)

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Détails bibliographiques

Titre : Studies on Renal Disorders (Hardback)
Éditeur : Humana Press Inc.
Date d'édition : 2010
Reliure : Hardback
Etat du livre : New

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Toshio Miyata
Edité par Humana Press Inc. (2010)
ISBN 10 : 1607618567 ISBN 13 : 9781607618560
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Description du livre Humana Press Inc., 2010. HRD. État : Used - Acceptable. Used Book. Shipped from UK in 4 to 14 days. Established seller since 2000. N° de réf. du libraire XJ-CRJ-14224772

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Editor-Toshio Miyata; Editor-Kai-Uwe Eckardt; Editor-Masaomi Nangaku
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Miyata, Toshio
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Edité par Humana Press Inc., United States (2010)
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Description du livre Humana Press Inc., United States, 2010. Hardback. État : New. Edition.. Language: English . Brand New Book. Conditions such as oxidative stress and hypoxia, which have a generalized impact on the oxygen metabolism, have been implicated in the genesis of kidney disease. This means that deepening our understanding of the pathobiology of oxygen metabolism in such diseases could be a fruitful path towards tangible clinical benefits. Studies in Renal Disorder collects reviews from leading researchers and clinical scientists working in exactly this field, providing an overview of the latest advances. The causal role of impaired oxygen metabolism in kidney disease has numerous clinical implications. It affects our understanding of the therapeutic benefits accruing from anti-hypertensive agents; the way we control hyperglycemia/hyperinsulinemia and hyperlipidemia; and our use of dietary approaches to the correction of obesity. The defensive mechanisms against oxidative stress, such as the Nrf2-Keap1 system, and hypoxia, such as the PHD-HIF system, have recently been explored in various cells, including kidney cells. These mechanisms include intracellular sensors for oxidative stress and hypoxia. This means that novel approaches targeting these sensors may offer clinical benefits in kidney disease in which oxidative stress and/or hypoxia is a final, common pathway. N° de réf. du libraire AAS9781607618560

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Edité par Humana Press Inc., United States (2010)
ISBN 10 : 1607618567 ISBN 13 : 9781607618560
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The Book Depository US
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Description du livre Humana Press Inc., United States, 2010. Hardback. État : New. Edition.. Language: English . Brand New Book. Conditions such as oxidative stress and hypoxia, which have a generalized impact on the oxygen metabolism, have been implicated in the genesis of kidney disease. This means that deepening our understanding of the pathobiology of oxygen metabolism in such diseases could be a fruitful path towards tangible clinical benefits. Studies in Renal Disorder collects reviews from leading researchers and clinical scientists working in exactly this field, providing an overview of the latest advances. The causal role of impaired oxygen metabolism in kidney disease has numerous clinical implications. It affects our understanding of the therapeutic benefits accruing from anti-hypertensive agents; the way we control hyperglycemia/hyperinsulinemia and hyperlipidemia; and our use of dietary approaches to the correction of obesity. The defensive mechanisms against oxidative stress, such as the Nrf2-Keap1 system, and hypoxia, such as the PHD-HIF system, have recently been explored in various cells, including kidney cells. These mechanisms include intracellular sensors for oxidative stress and hypoxia. This means that novel approaches targeting these sensors may offer clinical benefits in kidney disease in which oxidative stress and/or hypoxia is a final, common pathway. N° de réf. du libraire AAS9781607618560

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TOSHIO MIYATA
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Description du livre Humana Press, 2010. Hardback. État : NEW. 9781607618560 This listing is a new book, a title currently in-print which we order directly and immediately from the publisher. N° de réf. du libraire HTANDREE0308472

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Description du livre 2010. Hardcover. État : New. Hardcover. Conditions such as oxidative stress and hypoxia, which have a generalized impact on the oxygen metabolism, have been implicated in the genesis of kidney disease. This means th.Shipping may be from our Sydney, NSW warehouse or from our UK or US warehouse, depending on stock availability. 783 pages. 1.220. N° de réf. du libraire 9781607618560

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