Genome of PRSV pathotypes P (10317 nt) and W (10335 nt) from India were completely sequenced. Their genome architecture was similar to other 15 isloates from the rest of the world. Comparative sequence analysis revealed that pathotypes P and W shared high degree of sequence identity both at nucleotide (89%) and aminoacid (94%) levels. Sequence comparison of individual cistrons (except P1) also revealed similar trend. The P1 region was most divergent (upto 33%) and this could be attributed to geographical and host adaptation of PRSV. Cistron by cistron sequence comparison of PRSV pathotypes P and W from India with available 15 sequences and phylogenetic analysis based on full genome polyprotein revealed two distinct groupings of Asian and American isolates, although PRSV India clustered along with the American isolates. Putative recombination sites (24) were available through out the genomes, except in small 6K2 region. Maximum numbers of recombination sites were seen in 5′UTR and P1 region and has been speculated to be the most vulnerable region in shaping PRSV genome.
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Genome of PRSV pathotypes P (10317 nt) and W (10335 nt) from India were completely sequenced. Their genome architecture was similar to other 15 isloates from the rest of the world. Comparative sequence analysis revealed that pathotypes P and W shared high degree of sequence identity both at nucleotide (89%) and aminoacid (94%) levels. Sequence comparison of individual cistrons (except P1) also revealed similar trend. The P1 region was most divergent (upto 33%) and this could be attributed to geographical and host adaptation of PRSV. Cistron by cistron sequence comparison of PRSV pathotypes P and W from India with available 15 sequences and phylogenetic analysis based on full genome polyprotein revealed two distinct groupings of Asian and American isolates, although PRSV India clustered along with the American isolates. Putative recombination sites (24) were available through out the genomes, except in small 6K2 region. Maximum numbers of recombination sites were seen in 5′UTR and P1 region and has been speculated to be the most vulnerable region in shaping PRSV genome.
Dr. B. Parameswari, is presently working as Scientist (Plant Pathology) at Sugarcane Breeding Institute Regional Centre, Karnal. She completed her Ph.D from IARI, New Delhi. She is recipient of ICAR Jawaharlal Nehru award. Her research interests are in the areas of genomics, post transcriptional gene silencing and diagnosis of plant viruses.
Les informations fournies dans la section « A propos du livre » peuvent faire référence à une autre édition de ce titre.
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Taschenbuch. Etat : Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Genome of PRSV pathotypes P (10317 nt) and W (10335 nt) from India were completely sequenced. Their genome architecture was similar to other 15 isloates from the rest of the world. Comparative sequence analysis revealed that pathotypes P and W shared high degree of sequence identity both at nucleotide (89%) and aminoacid (94%) levels. Sequence comparison of individual cistrons (except P1) also revealed similar trend. The P1 region was most divergent (upto 33%) and this could be attributed to geographical and host adaptation of PRSV. Cistron by cistron sequence comparison of PRSV pathotypes P and W from India with available 15 sequences and phylogenetic analysis based on full genome polyprotein revealed two distinct groupings of Asian and American isolates, although PRSV India clustered along with the American isolates. Putative recombination sites (24) were available through out the genomes, except in small 6K2 region. Maximum numbers of recombination sites were seen in 5 UTR and P1 region and has been speculated to be the most vulnerable region in shaping PRSV genome. 180 pp. Englisch. N° de réf. du vendeur 9783659178313
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Taschenbuch. Etat : Neu. Genome organization of Papaya Ringspot Virus isolates from India | Complete genome organization and nucleotide sequence of PRSV pathotypes P and W | B. Parameswari (u. a.) | Taschenbuch | 180 S. | Englisch | 2012 | LAP LAMBERT Academic Publishing | EAN 9783659178313 | Verantwortliche Person für die EU: LAP Lambert Academic Publishing, Brivibas Gatve 197, 1039 RIGA, LETTLAND, customerservice[at]vdm-vsg[dot]de | Anbieter: preigu. N° de réf. du vendeur 106301054
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Taschenbuch. Etat : Neu. This item is printed on demand - Print on Demand Titel. Neuware -Genome of PRSV pathotypes P (10317 nt) and W (10335 nt) from India were completely sequenced. Their genome architecture was similar to other 15 isloates from the rest of the world. Comparative sequence analysis revealed that pathotypes P and W shared high degree of sequence identity both at nucleotide (89%) and aminoacid (94%) levels. Sequence comparison of individual cistrons (except P1) also revealed similar trend. The P1 region was most divergent (upto 33%) and this could be attributed to geographical and host adaptation of PRSV. Cistron by cistron sequence comparison of PRSV pathotypes P and W from India with available 15 sequences and phylogenetic analysis based on full genome polyprotein revealed two distinct groupings of Asian and American isolates, although PRSV India clustered along with the American isolates. Putative recombination sites (24) were available through out the genomes, except in small 6K2 region. Maximum numbers of recombination sites were seen in 5¿UTR and P1 region and has been speculated to be the most vulnerable region in shaping PRSV genome.VDM Verlag, Dudweiler Landstraße 99, 66123 Saarbrücken 180 pp. Englisch. N° de réf. du vendeur 9783659178313
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Taschenbuch. Etat : Neu. nach der Bestellung gedruckt Neuware - Printed after ordering - Genome of PRSV pathotypes P (10317 nt) and W (10335 nt) from India were completely sequenced. Their genome architecture was similar to other 15 isloates from the rest of the world. Comparative sequence analysis revealed that pathotypes P and W shared high degree of sequence identity both at nucleotide (89%) and aminoacid (94%) levels. Sequence comparison of individual cistrons (except P1) also revealed similar trend. The P1 region was most divergent (upto 33%) and this could be attributed to geographical and host adaptation of PRSV. Cistron by cistron sequence comparison of PRSV pathotypes P and W from India with available 15 sequences and phylogenetic analysis based on full genome polyprotein revealed two distinct groupings of Asian and American isolates, although PRSV India clustered along with the American isolates. Putative recombination sites (24) were available through out the genomes, except in small 6K2 region. Maximum numbers of recombination sites were seen in 5 UTR and P1 region and has been speculated to be the most vulnerable region in shaping PRSV genome. N° de réf. du vendeur 9783659178313
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