Current Progress in the Understanding of Secondary Brain Damage from Trauma and Ischemia. Acta Neurochirurgica, Supplement 73.

Editors: A. Baethmann, N. Plesnila, F. Ringel, J. Eriskat:

ISBN 10: 3211209328 / ISBN 13: 9783211209325

Edité par Wien/New York, Springer, 1999

A propos de cet article

zahlreiche Abb., 116 Seiten, Zustand: sehr gutes Exemplar, minimale Gebrauchsspuren; aus der Bibliothek eines namhaften Neurologen! Sprache: Englisch Gewicht in Gramm: 1100 28,5 x 21,0 cm, gebundene Ausgabe, Hardcover/Pappeinband.

N° de réf. du vendeur 29173

Détails bibliographiques

• Titre: Current Progress in the Understanding of Secondary Brain Damage from Trauma and Ischemia. Acta Neurochirurgica, Supplement 73.
• Éditeur: Wien/New York, Springer
• Date de parution: 1999
• Langue: anglais
• ISBN à 10 chiffres: 3211209328
• ISBN à 13 chiffres: 9783211209325
• Reliure: Couverture rigide
• Poids: 1100 grammes
• Catalogues du vendeur:
  • Naturwissenschaft&Medizin

A propos de ce titre

Synopsis

The international interdisciplinary gathering of top of secondary brain damage in brain trauma, as ac level clinical and laboratory scientists in Mauls, Italy knowledged from the beginning of these workshops in has developed from its beginning in 1984into a fruitful Mauls, the significance of inflammation is all but clear. tradition where worldwide experts active and knowl Although inflammatory phenomena are seen in trauma edgeable in cerebral ischemia and trauma convene for and ischemia ofthe brain, as activation ofwhite blood update and exchanges of their most recent clinical and cells with emigration into the tissue presumably en experimental findings and concepts. These meetings hancing damage, inflammatory cells may have benefi have, of course, experienced shifts in emphasis from cial properties as well. Thechapter on the Janus-faceof the past until now, corresponding to the most actual inflammation isanalyzing this ambiguity. developments, which were fascinating clinicians and The exploration of novel cell-biological mechanisms laboratory scientists alike. The current Supplement of on a molecular or more systemic basis causing apop Acta Neurochirurgica is an example in case. Its virtue totic cell death, inflammation, or regeneration, provide as before is that authors contribute articles in a review useful objectives for therapeutical interventions ex like manner on their own field of research, according pected to be more specific than the present treatment to the platform presentations at the meeting as indis modalities."