Evaluation of Outcome in Thymectomized Myasthenia Gravis Patient

Mohammad Ata Ullah (u. a.)

ISBN 10: 6139851246 ISBN 13: 9786139851249

Edit� par LAP LAMBERT Academic Publishing, 2018

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Titre : Evaluation of Outcome in Thymectomized ...
�diteur : LAP LAMBERT Academic Publishing
Date d'�dition : 2018
Reliure : Taschenbuch
Etat : Neu

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Evaluation of Outcome in Thymectomized Myasthenia Gravis Patient

Mohammad Ata Ullah|Asit Baran Adhikary|Md Rezwanul Hoque

Edit� par LAP LAMBERT Academic Publishing, 2018

ISBN 10 : 6139851246 ISBN 13 : 9786139851249

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Etat : New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Autor/Autorin: Ata Ullah MohammadDr Mohammad Ata Ullah from a reputed muslim family of Noakhali, Bangladesh. Working at now BSMMU, Dhaka. Interested in research.Myasthenia gravis is an autoimmune disease characterized by a fluctuating weakness . N� de r�f. du vendeur 385874300

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Evaluation of Outcome in Thymectomized Myasthenia Gravis Patient

Mohammad Ata Ullah

Edit� par LAP LAMBERT Academic Publishing Jun 2018, 2018

ISBN 10 : 6139851246 ISBN 13 : 9786139851249

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Taschenbuch. Etat : Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Myasthenia gravis is an autoimmune disease characterized by a fluctuating weakness of voluntary muscles (Busch et al. 1996). The weakness of myasthenic patients is due to an antibody-mediated autoimmune attack against acetylcholine receptor at neuromuscular junction (Urschel et al. 1998). Normally impulses travel down the nerve and nerve endings release a substance called acetylcholine. Acetylcholine binds or attaches to receptor on the muscle and makes the muscle contract. The strength of the contraction depends on how much acetylcholine the muscles receive. In myasthenia gravis, Immune system produces antibodies that block or destroy many of the receptor sites for acetylcholine in muscle. Because there are fewer receptor sites, the muscles get fewer nerve signals and become weak. In another form of MG, Immune system produces antibodies to the muscle specific tyrosine kinase protein. 108 pp. Englisch. N� de r�f. du vendeur 9786139851249

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Evaluation of Outcome in Thymectomized Myasthenia Gravis Patient

Mohammad Ata Ullah

Edit� par LAP LAMBERT Academic Publishing Jun 2018, 2018

ISBN 10 : 6139851246 ISBN 13 : 9786139851249

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Taschenbuch. Etat : Neu. This item is printed on demand - Print on Demand Titel. Neuware -Myasthenia gravis is an autoimmune disease characterized by a fluctuating weakness of voluntary muscles (Busch et al. 1996). The weakness of myasthenic patients is due to an antibody-mediated autoimmune attack against acetylcholine receptor at neuromuscular junction (Urschel et al. 1998). Normally impulses travel down the nerve and nerve endings release a substance called acetylcholine. Acetylcholine binds or attaches to receptor on the muscle and makes the muscle contract. The strength of the contraction depends on how much acetylcholine the muscles receive. In myasthenia gravis, Immune system produces antibodies that block or destroy many of the receptor sites for acetylcholine in muscle. Because there are fewer receptor sites, the muscles get fewer nerve signals and become weak. In another form of MG, Immune system produces antibodies to the muscle specific tyrosine kinase protein.VDM Verlag, Dudweiler Landstra�e 99, 66123 Saarbr�cken 108 pp. Englisch. N� de r�f. du vendeur 9786139851249

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Evaluation of Outcome in Thymectomized Myasthenia Gravis Patient

Mohammad Ata Ullah

Edit� par LAP LAMBERT Academic Publishing, 2018

ISBN 10 : 6139851246 ISBN 13 : 9786139851249

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Taschenbuch. Etat : Neu. nach der Bestellung gedruckt Neuware - Printed after ordering - Myasthenia gravis is an autoimmune disease characterized by a fluctuating weakness of voluntary muscles (Busch et al. 1996). The weakness of myasthenic patients is due to an antibody-mediated autoimmune attack against acetylcholine receptor at neuromuscular junction (Urschel et al. 1998). Normally impulses travel down the nerve and nerve endings release a substance called acetylcholine. Acetylcholine binds or attaches to receptor on the muscle and makes the muscle contract. The strength of the contraction depends on how much acetylcholine the muscles receive. In myasthenia gravis, Immune system produces antibodies that block or destroy many of the receptor sites for acetylcholine in muscle. Because there are fewer receptor sites, the muscles get fewer nerve signals and become weak. In another form of MG, Immune system produces antibodies to the muscle specific tyrosine kinase protein. N� de r�f. du vendeur 9786139851249

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