Pathogenicity Islands and the Evolution of Pathogenic Microbes: Volume I: 264/1 (Current Topics in Microbiology and Immunology, 264/1) [Hardcover] Hacker, J. and Kaper, J.B.

ISBN 10: 3540426817 / ISBN 13: 9783540426813

Edité par Springer, 2002

A propos de cet article

Used, some outer edges and corners have minor scuffs, cover has light scratches, book content is in very good condition.

N° de réf. du vendeur 090440-6

Détails bibliographiques

• Titre: Pathogenicity Islands and the Evolution of Pathogenic Microbes: Volume I: 264/1 (Current Topics in Microbiology and Immunology, 264/1) [Hardcover] Hacker, J. and Kaper, J.B.
• Éditeur: Springer
• Date de parution: 2002
• Langue: anglais
• ISBN à 10 chiffres: 3540426817
• ISBN à 13 chiffres: 9783540426813
• Reliure: Couverture rigide
• État de l'article: Very Good

A propos de ce titre

Synopsis

It has been known for a number of years that not only pathogenicity islands but also plasmids and bacteriophages are able to carry genes whose products are involved in pathogenic processes. Accordingly, such elements and their products play an important role in pathogenesis due to the intestinal E. coli as well due to Shigellae. Another interesting aspect which is reflected in different articles is that genomes evolve by acquisition of new pieces of DNA following gene transfer, but also by genome reduction. Different mechanisms include the deletion of sequences or the elimination of functions by the accumulation of point mutations or rearrangements.

Présentation de l'éditeur

It has been known for a number of years that not only pathogenicity islands but also plasmids and bacteriophages are able to carry genes whose products are involved in pathogenic processes. Accordingly, such elements and their products play an important role in pathogenesis due to the intestinal E. coli as well to Shigellae. Another interesting aspect which is reflected in different articles is that genomes evolve by acquisition of new pieces of DNA following gene transfer, but also by genome reduction. Different mechanisms include the deletion of sequences or the elimination of functions by the accumulation of point mutations or rearrangements.