J j mueller eberhard (64 résultats)

Broschiert. Etat : Gut. 335 Seiten; Das hier angebotene Buch stammt aus einer teilaufgelösten Bibliothek und kann die entsprechenden Kennzeichnungen aufweisen (Rückenschild, Instituts-Stempel.); der Buchzustand ist ansonsten ordentlich und dem Alter entsprechend gut. Neben dem oben aufgeführten Aufsatz befinden sich auch weitere Beiträge auch anderer Autoren in dem Werk. In ENGLISCHER Sprache. Sprache: Englisch Gewicht in Gramm: 560.…

Etat : good. Befriedigend/Good: Durchschnittlich erhaltenes Buch bzw. Schutzumschlag mit Gebrauchsspuren, aber vollständigen Seiten. / Describes the average WORN book or dust jacket that has all the pages present.

Etat : Gut. Softcovereinband, 152 Seiten, gutes Exemplar. 814-hw23237.

Softcover. 236 pages. Clean, good binding, cover has bending. Illustrated with 44 figures and 39 tables. Deals with pharmacological research and pharmacology natural history as well as developments etc. Papers by various persons in the field; 1980-01-01.

Hardcover. Etat : gut. Auflage: 1., Inklusive fabilon. Im Unternehmen werden Unternehmer, Führungskräfte und deren Berater ständig mit Haftungs- und Risikofragen konfrontiert. Besonders gefährlich sind die Risiken, die nicht oder nicht rechtzeitig erkannt werden. So führen kritische Ereignisse immer häufiger zu Schadensersatzansprüchen gegen das Unternehmen und die handelnden Personen. Die Haftungssituation für Unternehmen wird nicht nur durch die sich immer schneller ändernde Gesetzgebung spürbar verschärft. Zunehmend erhöht neues nationales und supranationales Recht (z. B. Corporate Governance Kodex) das Risiko für die Handelnden. "Haftungsgefahren im Unternehmen" zeigt nicht nur die Risiken auf, sondern hilft auch aktiv, sie zu vermeiden. Das neue Handbuch richtet sich dabei konsequent nach den Bedürfnissen der Verantwortlichen in den Unternehmen. Die Autoren verzichten auf "Fachchinesisch" - sie schreiben Klartext. Zahlreiche Praxishinweise und Beispiele bieten Lösungen und Anregungen zur praktischen Umsetzung. Die einzelnen Haftungs- und Risikobereiche sind übersichtlich als Stichwort-ABC aufgefächert. Das garantiert ein schnelles Auffinden der Problembereiche. Ein weiterer Vorteil: Das Handbuch liefert praxisgerechtes Know-how für alle Rechtsformen im Unternehmensbereich. Inhaltsübersicht 1. Kapitel: Haftungsprobleme im Unternehmen - Ein Überblick A. Erhöhte Haftungsrisiken in Unternehmen B. Grundlagen des deutschen Haftungsrechts C. Blick über die Grenze - Managerhaftung in Auslandsgesellschaften 2. Kapitel: Haftungslexikon (ca. 200 Stichworte - von "Abfallentsorgung" bis "Zeugnis") 3. Kapitel: Konsequenzen in der Praxis A. Risikomanagement B. Zusammenarbeit mit Beratern C. Verhalten im Schadensfall 4. Kapitel: Regress - Beraterhaftung Überarbeitung Ralf-Wolfgang Ahlmann, Pia Maria Aigner, Helmut Ammann, Frank Michael Bauer, Anne Baum-Rudischhauser, Frank J. Bernardi, Franz-Bernd Daum, Bernd Ebel, Harald Ehlers, Christoph Eipper, Rainald Enders, Claus Fleischmann, Roland Geres, Horst Grätz, Jörg Greck, Friederike Heidmeier, Joachim Hermes, Susanne Hierl, Andreas Hilge, Heike Hinrichs, Hans Reinold Horst, Anja Hucke, Heiko Huß, Ralf Josten, Dirk Kenntner, Jürgen Kozielsky, Michael Krings, Christoph Landscheidt, Volker Lang, Konrad Löcherbach, Andreas Marschner, Reinhold Mauer, Regina Michalke, Andreas Mraz, Norbert Müller, Michael Pilz, Jörg Reichelsdorfer, Kornelia Reinke, Ulrich Rieck, Rainer Schaaf, Uwe J. Scherf, Andreas Schiele, Michael Schmalholz, Harald Schumm, Matthias Spirolke, Ralf Stölzel, Eberhard Temme, Thomas Tetzner, von Schubert Michael, Nikolaus Weber, Klaus Wolf, Martin Wörlein Sprache deutsch Einbandart gebunden Recht Steuern Privatrecht Bürgerliches Recht e Aufsichtsratshaftung Außenhaftung Beiratshaftung Deutschland Durchgriffshaftung Geschäftsführerhaftung Haftung Rechtlich Bürgerliches Recht Zivilprozessrecht BGB Managerhaftung Recht Privatrecht Unternehmen Unternehmenshaftung Unternehmerhaftung Vorstandshaftung ISBN-10 3-482-52671-0 3482526710 ISBN-13 978-3-482-52671-8 9783482526718 Haftungsgefahren im Unternehmen. Ein Handbuch für Führungskräfte und deren Berater [Gebundene Ausgabe] von K. Jan Schiffer (Herausgeber), Christian Rödl (Herausgeber), Eberhard Rott (Herausgeber), Ralf-Wolfgang Ahlmann (Herausgeber), Pia Maria Aigner (Herausgeber), Helmut Ammann (Herausgeber), Frank Michael Bauer (Herausgeber), Anne Baum-Rudischhauser (Herausgeber), Frank J. Bernardi (Herausgeber), Franz-Bernd Daum (Herausgeber), Bernd Ebel (Herausgeber), Harald Ehlers (Herausgeber), Christoph Eipper (Herausgeber), Rainald Enders (Herausgeber), Claus Fleischmann (Herausgeber), Roland Geres (Herausgeber), Horst Grätz (Herausgeber), Jörg Greck (Herausgeber), Friederike Heidmeier (Herausgeber), Joachim Hermes (Herausgeber), Susanne Hierl (Herausgeber), Andreas Hilge (Herausgeber), Heike Hinrichs (Herausgeber), Hans Reinold Horst (Herausgeber), Anja Hucke (Herausgeber), Heiko Huß (Herausgeber), Ralf Josten (Herausgeber), Dirk Kenntner (Herausgeber), Jürgen Kozielsky (Herausgeber), Michael Krings (Herausgeber), Christoph Landscheidt (Herausgeber), Volker Lang (Herausgeber), Konrad Löcherbach (Herausgeber), Andreas Marschner (Herausgeber), Reinhold Mauer (Herausgeber), Regina Michalke (Herausgeber), Andreas Mraz (Herausgeber), Norbert Müller (Herausgeber), Michael Pilz (Herausgeber), Jörg Reichelsdorfer (Herausgeber), Kornelia Reinke (Herausgeber), Ulrich Rieck (Herausgeber), Rainer Schaaf (Herausgeber), Uwe J. Scherf (Herausgeber), Andreas Schiele (Herausgeber), Michael Schmalholz (Herausgeber), Harald Schumm (Editor), Matthias Spirolke (Editor), Ralf Stölzel (Editor), Eberhard Temme (Editor), Thomas Tetzner (Editor), Michael Schubert von (Editor), Nikolaus Weber (Editor), Klaus Wolf (Editor), Martin Wörlein (Editor) In deutscher Sprache. 816 pages. 24,4 x 17,8 x 4,8 cm.…

Opd.m.OU. 191 S. : überw. Ill. ; 31 cm umschlag mit Läsuren Sprache: Deutsch Gewicht in Gramm: 1368.

gebundene Ausgabe. Etat : Gut. 8005 Seiten; duchgehene Zählung; Die hier angebotenen Bände stammen aus einer teilaufgelösten Bibliothek und weisen die entsprechenden Kennzeichnungen auf (Rückenschild, Instituts-Stempel.); der Buchzustand ist ansonsten ordentlich und dem Alter entsprechend gut. Text in ENGLISCHER Sprache; KOMPLETTPREIS für 2 Bände; bei Versand außerhalb der EU erfragen Sie bitte zuerst die Versandkosten; Sprache: Englisch Gewicht in Gramm: 2900.…

Etat : New.

Etat : New. In English.

Etat : New. In English.

Etat : New. In English.

Etat : New. In English.

Kartoniert / Broschiert. Etat : New. Peter A. Miescher The skin has always been one of the major targets to clinicians interested in immunology. Initially, the main focus was centred on allergic conditions such as contact hypersensitivity and urticeria. Gradually, the spectrum of immunological.

Etat : New. Howard C. Thomas In normal subjects the regulatory apparatus of the immune system permits responses to foreign antigens but suppresses those directed to self components. Autoimmune disease occurs as a failure ofthis system either as a result of a primary .

Etat : New. The discovery of specifically acquired immunity which followed the major contributions of Louis Pasteur completely over-shadowed the first studies of the host s natural resistance. Later, the exquisite sensitivity and precision of antigen-antibody reactions.

Kartoniert / Broschiert. Etat : New. The subject of immune deficiency has become of special importance for two reasons. First, conditions with well defined defects in the immune system could be analyzed as experiments of nature in terms of finding out the accurate biological relevance of the.

Etat : New. pp. 196.

Taschenbuch. Etat : Neu. Druck auf Anfrage Neuware - Printed after ordering - Howard C. Thomas In normal subjects the regulatory apparatus of the immune system permits responses to foreign antigens but suppresses those directed to 'self' components. Autoimmune disease occurs as a failure ofthis system either as a result of a primary defect in the regulatory apparatus (primary autoimmunization) or because of a change in the antigenicity of the tissues (secondary autoimmunization). Autoaggressive reactions are characterised by the presence of autoantibodies. When these are directed to membrane displayed antigens (Fig. 1) they are probably of importance in the lysis of hepatocytes. Those directed to cytoplasmic antigens may be useful diagnostically but are of unknown pathogenic significance. When no extrinsic aetiological factor can be identified, the process is assumed to be the result of a failure ofthe regulatory system, allowing the spontaneous expansion of a clone of autoreactive lymphocytes. The defect may be generalised or specific to certain groups of self-antigens and thus the autoimmune disease may be either multi- or unisystemic. The recent development of techniques to enumerate and measure the functional activity of the suppressor lymphocytes which control the effector limbs of the immune system has enabled investigators to test whether the various purported autoimmune diseases do have as their basis a generalised defect in immunoregu lation. Assessment of antigen-specific immunoregulatory function is, however, not yet readily available. liver Membrane I Antigen (LIM) I Liver I HLA, A, B, C, Sensitisation to Specific -;::!IIIL. .…

Taschenbuch. Etat : Neu. Druck auf Anfrage Neuware - Printed after ordering - Peter A. Miescher The skin has always been one of the major targets to clinicians interested in immunology. Initially, the main focus was centred on allergic conditions such as contact hypersensitivity and urticeria. Gradually, the spectrum of immunological conditions involving the skin increased, encompassing diseases such as Dermatitis Herpetiformis, Erythema Muitiforme, Cutaneous Bullous Disorders and Photo toxic Reactions. It has been the intention of the editors of Springer Seminars in Immu nopathology to cover the broad spectrum of immune phenomena encountered in dermatological diseases with special emphasis to newer developments in Immunopathology. Dr. 1. N. Gigli has managed to assemble a series of important contributions within two issues of the Journal which are of such an interest to dermatologists that we decided to publish this topic as a separate volume for all dermatologists who do not have access to Seminars in Immunopathology. 3 Immunologically Mediated Epidermal Cell Injury Richard D. Sontheimer\* and James N. Gilliam Division of Dermatology, University of Texas Health Science Center, 5323 Harry Hines Boulevard, Dallas, Texas 75235, USA Introduction The epidermis is a functional unit of different cell types that has the critical role of protecting man from his environment. The three major cell types that make up the epidermis (keratinocytes, melanocytes, and Langerhans cells) serve different protective functions. The vast majority of epidermal cells are keratinocytes (Fig. 1). …

Taschenbuch. Etat : Neu. Druck auf Anfrage Neuware - Printed after ordering - The subject of immune deficiency has become of special importance for two reasons. First, conditions with well defined defects in the immune system could be analyzed as 'experiments of nature' in terms of finding out the accurate biological relevance of the defective link in the immune system. Secondly, the recognition of immune deficiency states has become important in order to provide the patients with the treatment necessary to remedy these defects. With regard to immune deficiency states in patients, these have been instrumental as 'experiments of nature' in the revelation by Drs. Good and Cooper and their associates of the two-component structure of the immune system, a discovery which can be consi dered as a major breakthrough in the history of immunopathology. Today's research allows us to go far beyond this basic two component structure with the assessment of disorders affecting either cell to cell interactions or regarding subsets of lymphocyte populations. Furthermore, the association of immune deficiency with distinct enzymatic defects of purine metabolism is opening the door to the molecular level of immune deficiency. Dr. Cooper and Dr. Lawton have succeeded in obtaining the collaboration of the leaders in the field of immune deficiency. In view of the importance of their contributions in scientific and clinical terms, we decided to prepare a book version of the two issues of Seminars in Immunopathology devoted to this subject.…

Taschenbuch. Etat : Neu. Druck auf Anfrage Neuware - Printed after ordering - Activation of the complement system gives rise to a number of molecular species which can interact with host-derived cells and regulate their function. This interaction is mediated through distinct cell surface complement receptors, and receptor engagement produces biologic responses which can either modulate host defense reactions or enhance inflammation. Although the first complement receptor was recognized more than 30 years ago [80J, detailed biochemical information concerning the receptors has only recently become available. Currently, eight distinct complement receptors are recognized. Five receptors (CRl, CR2, CR3, C3a receptor, and C3e receptor) react with various regions on C3 while the other receptors display specificity for Clq, C5a, or Factor H. This chapter focusses on the chemistry of the various ligands and receptors and discusses the biologic activities which arise as a result of receptor-ligand interaction. II. CIQ Receptor A. The Ligand Clq is the recognition molecule of the classical complement pathway [reviewed in 20, 130]. In serum, it is part of a calcium-dependent penta molecular complex containing one molecule ofClq and two molecules each of the pro enzymes Clr and CIs. During classical pathway activation, the macromolecular complex becomes associated with the activator through the Clq subcomponent. Bound Clq undergoes a conformational change and induces the proteolytic autoactivation of Clr which in turn effects the proteolytic activation of CIs. This process is controlled by the Cl inhibitor (ClINH) which binds irreversibly to activated Clr and CIs, and inhibits their enzymatic activities.…

Etat : New. pp. 196.

Paperback. Etat : Brand New. illustrated edition. 196 pages. 9.61x6.54x0.55 inches. In Stock.

Paperback. Etat : Brand New. 1st edition. 196 pages. 9.61x6.69x0.45 inches. In Stock.

Etat : New. pp. 484.

Etat : New. pp. 220.

Paperback. Etat : Brand New. illustrated edition. 220 pages. 9.61x6.69x0.50 inches. In Stock.

Paperback. Etat : Brand New. 1st edition. 480 pages. 9.61x6.69x1.09 inches. In Stock.

Etat : Good. Your purchase helps support Sri Lankan Children's Charity 'The Rainbow Centre'. Ex-library, so some stamps and wear, but in good overall condition. Our donations to The Rainbow Centre have helped provide an education and a safe haven to hundreds of children who live in appalling conditions.…

Etat : Sehr gut. Zustand: Sehr gut | Seiten: 484 | Sprache: Englisch | Produktart: Bücher | Activation of the complement system gives rise to a number of molecular species which can interact with host-derived cells and regulate their function. This interaction is mediated through distinct cell surface complement receptors, and receptor engagement produces biologic responses which can either modulate host defense reactions or enhance inflammation. Although the first complement receptor was recognized more than 30 years ago [80J, detailed biochemical information concerning the receptors has only recently become available. Currently, eight distinct complement receptors are recognized. Five receptors (CRl, CR2, CR3, C3a receptor, and C3e receptor) react with various regions on C3 while the other receptors display specificity for Clq, C5a, or Factor H. This chapter focusses on the chemistry of the various ligands and receptors and discusses the biologic activities which arise as a result of receptor-ligand interaction. II. CIQ Receptor A. The Ligand Clq is the recognition molecule of the classical complement pathway [reviewed in 20, 130]. In serum, it is part of a calcium-dependent penta molecular complex containing one molecule ofClq and two molecules each of the pro enzymes Clr and CIs. During classical pathway activation, the macromolecular complex becomes associated with the activator through the Clq subcomponent. Bound Clq undergoes a conformational change and induces the proteolytic autoactivation of Clr which in turn effects the proteolytic activation of CIs. This process is controlled by the Cl inhibitor (ClINH) which binds irreversibly to activated Clr and CIs, and inhibits their enzymatic activities.…